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Inflammation effects on human organs

Inflammation effects

Inflammation effects on human organs are real and can be treated professionally

Inflammation effects on human organs: Damages caused by inflammation

Some of the health conditions affecting our life are known to us yet we often don’t notice them easily. Take for instance all the problem that are associated with chronic inflammation effects; many of us do not take notice of this until we experience an injury either by way of swellings, redness and pain flood that affects the area. But even with this indication, when we opted for treatment our focus is always on the pain and not the internal healing process. Inflammation can damage organs and cause disease, but its interference in the brain causes two common disorders. We are therefore going to be discussing the effects of inflammation on the body organs for a better understanding of the body’s vulnerability to this complication. Doctor Dalal Akoury explains that ordinarily, your body’s defense mechanism kicks in, and you don’t have to think too much about inflammation because it is always there when you need it.

However, even though this is so, inflammation might also occur when you don’t need it, and if it does, it can cause problems. In some people, inflammation is present at very low levels. It is no wonder that many experts now believe that this type of low-grade, chronic inflammation is responsible for a litany of chronic conditions. And according to the experts from AWAREmed Health and wellness Resource Center under the able leadership of doctor Dalal Akoury MD, when inflammation extends to the pancreas the affected person is at risk of getting diabetes. When it spreads to the immune system, you get cancer. And like cancer, this type of inflammation grows and spreads, damaging organs and causing all kinds of trouble. We will be focusing on this for a while but in the meantime, if you have any concern about this problem, doctor Akoury is always available for you and will help you in the best and professional manner.

Inflammation effects on human organs: Weakened signals

The speed with which inflammation spreads does not only cause damages to your organs but also interferes with your brain’s signals and to the rest of your body systems. With such many conditions, the trouble starts in the hypothalamus which is the command center of the brain. The command center then receives all these hormonal inputs that inform you when you’re tired or when to eat and what to do with those calories.

When you have inflammation, a protein called pro-inflammatory cytokines distorts those hormonal signals. As a result, inflammation might be causing conditions such as depression and obesity, which were correlated through research long before inflammation became a suspected culprit. Nonetheless, when it comes to depression, those cytokines communicate with the brain to induce different depressive symptoms, such as sad mood, fatigue, altered sleep and social-behavioral withdrawal. Inflammation also drives obesity in a similar manner.

Inflammation effects on human organs: Damages caused by inflammation

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Cellular body inflammation causes obesity

Cellular body inflammation

Cellular body inflammation causes obesity can all be addressed beginning with change of life style

Cellular body inflammation causes obesity: Health complications attached

A common theme that links many diseases and chronic illness is uncontrolled cellular body inflammation. It is a factor with most diseases including cardiovascular disease, diabetes, cancer, arthritis and many autoimmune-related conditions. And from the expert desk at AWAREmed health and wellness resource center under the leadership of doctor Dalal Akoury MD, it will interest you to note that recently obesity has recently been added to this group of diseases because it is equally presenting a low grade inflammatory response within many of the body’s tissues, which cause deleterious effects leading to the development of cardiovascular and metabolic disease.

It is well known that being overweight is detrimental to one’s health, but until recently the known mechanisms were limited. Scientists over the last decade have started to unravel the mystery of why obesity leads to premature death. Although there is still much to learn, it is valuable to comprehend the known effects of chronic inflammation, as the prevalence of obesity continues to be a rising problem among the American population, particularly in children.

Cellular body inflammation causes obesity: Chronic Inflammation

Inflammation is, by design, a protective response leading to the repair of tissue. When inflammation becomes chronic, as is the case with obesity, chemical mediators, derived from different cellular activities, change in dynamics causing a progressive state of decline. Fat cells are now considered an immune organ that secretes numerous immune modulating chemicals. Visceral fat, in particular, is associated with the low grade inflammation that seems to be a contributing pathologic feature for metabolic disease through insulin resistance and the promotion of atherosclerotic build-up in circulatory vessels.

When high levels of visceral fat are combined with physical inactivity, over nutrition, and advancement in age, the effect becomes more pronounced. Visceral fat is highly metabolic and contributes to cytokine hyperactivity. Adipokines secreted from fat tissue influence the metabolic process and contribute to proper function. The consequent low grade inflammation associated with obesity causes disturbance in the secretion and function of adipokines. Research has identified changes in adiponectin, leptin, and resisting that exhibit harmful effects upon the body in obese individuals.  Adiponectin is an antiatherogenic agent, meaning it helps prevent the development of atheroschlerotic plaque in blood vessels and slows the progression of atherosclerosis in coronary vessels. It does this by acting directly upon the vessel wall, inhibiting adhesive molecules from contributing to plaque formation and acts as a blocking agent to the formation of foam cells.

Finally, take note that in the skeletal muscle and the liver, adiponectin serves to promote insulin sensitivity and a positive blood lipid profile. Visceral adiposity reduces adiponectin concentrations. Lowering the adiponectin concentrations lessens the cardio protective effect, leading to increased cardiovascular risk. You may have realized that in this discussion of cellular body inflammation and how it causes obesity, a lot of medical terminologies are applied which may not make sense to many lay people. In appreciating that fact and to help you take good precautions about obesity and overweight, we want to encourage you to schedule an appointment with doctor Dalal Akoury not for a one on one simplicity discussions that will address all your concerns and have then addressed professionally.

Cellular body inflammation causes obesity: Health complications attached

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Inflammation effects on human organs

Other inflammation effects

Other inflammation effects on human organs can be dealt with using preventive treatment approach

Other inflammation effects on human organs: Metabolic syndrome

Inflammation has recently been linked with metabolic syndrome, which is a cluster of symptoms that raise your risk for chronic conditions such as heart disease, stroke and diabetes. Those symptoms include hypertension, low levels of HDL (good) cholesterol, above-normal blood glucose and triglyceride levels, and a large waist. Most people with metabolic syndrome are obese and inactive, so these symptoms have become associated with obesity. However, many obese people don’t have those symptoms. They fall into a category called metabolically healthy obese. What that means explains doctor Dalal Akoury is that they don’t look very good in a swimsuit, but if you look at their blood, they’re in pretty good shape. This is probably because they don’t have chronic inflammation. Meaning that among other inflammation effects, we may not say that obesity causes inflammation, but we can comfortably say that inflammation drives obesity.

Other inflammation effects on human organs: Depression

Doctor Akoury says that inflammation may drive depression in a similar fashion. The links between inflammation and depression are complex and we are just beginning to understand how they affect each other. Though it is possible that depression may lead to changes that cause inflammation, experimental studies have shown that increases in inflammation can promote depression.

According to Canada’s Centre for Addiction and Mental Health findings, it was established that people suffering from depression had 30 percent higher levels of inflammation in the brain. This was one of the first studies to show concretely that inflammation is present in depression even when other conditions are absent. Moreover depression is still complex and unlikely to be caused by inflammation alone just like obesity.

When I started my career in the medical profession, and like many of my colleagues, I generally thought about depression as a psychiatric disorder or a mental health problem and I believe that many people still think of it that way. But nevertheless what I have come to realize is that depression is also a disorder with deep biological roots. It is as much of a physical issue as a mental one, and we should start talking about it that way. Like for instance, if you were to compare depression to a more physical illness. When you have the flu, your body’s immune system springs into action to fight it off, including increasing inflammation. In addition to the nausea and fever, you also have symptoms of depression: fatigue, aches, mental fog and an unwillingness to leave the house. In a contagious illness, those symptoms help us rest and avoid spreading the disease, but in depression they’re nothing but trouble.

Other inflammation effects on human organs: Minimizing inflammation

Tests that indicate your levels of inflammation are available, but they can be expensive and aren’t always conclusive. They almost certainly aren’t covered by insurance unless you have an autoimmune disorder. It therefore means that you may never know your true levels of inflammation; however you can still do your part to control it. Controlling inflammation is a balancing act. You need some, but not too much. By far, the best drug we have for this is diet.

Certain staples of the recent American diet promote high levels of inflammation. The two largest culprits are omega-6 fatty acids and insulin spikes brought on by consuming starchy carbs. The combination of insulin and omega-6 fatty acids is like a match and gasoline you get an explosion of this low-level inflammation.

To reduce it, the best foods are those with omega-3 fatty acids and high levels of polyphenols, the compounds in fruits and vegetables that give them color. These good fats and colorful fruits and vegetables, along with lean protein, make up the majority of his anti-inflammatory meal plan. Along with diet, a healthy lifestyle and stress reduction techniques help reduce levels of inflammation, especially in the brain. It is also important to maintain a healthy diet, stay active, keep a regular sleep schedule and limit the psychological stress we experience. And in all these ensure to consult with the experts at AWAREmed Health and Wellness Resource Center for professional advices from time to time.

Other inflammation effects on human organs: Metabolic syndrome

 

 

 

 

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Inflammation effects on cancer patients

Inflammation effects

Inflammation effects on cancer patients can be very painful and the time for change is now

Inflammation effects on cancer patients: Benefits of good nutrition

The meaning of inflammation can be understood in two different ways depending on how it affects the body. Like for instance inflammation effects could be defined as a localized physical condition in which part of the body becomes reddened, swollen, hot, and often painful, especially as a reaction to injury or infection. It could also be defined as the body’s attempt at self-protection; the aim being to remove harmful stimuli, including damaged cells, irritants, or pathogens – and begin the healing process. What this means is that when anything harmful or irritating affects any part of our bodies, there will be always a biological response to try to remove it.

The understanding of that is that the signs and symptoms of inflammation effects, and specifically the acute inflammation, show that the body is trying to heal itself. Therefore inflammation effects does not necessarily mean infection, even when an infection causes inflammation. Infection itself is caused by a bacterium, virus or fungus, while inflammation is the body’s response to it. With that elaborate understanding of what inflammation is, the a million dollar question would be what does it have to do with cancer? In other words what are some of the inflammation effects on cancer patients? Keep reading and in a little while doctor Dalal Akoury MD and founder of AWAREmed Health and Wellness Resource Center will be giving us the finer details.

Prolonged inflammation

The truth of the matter is that irrespective of the definition, prolonged inflammation can cause real damage to your body’s healthy cells and tissue thereby weakening your immune system. It is this state of weakness in the immunity that increases ones risk of contracting the cancer diseases. But like we had indicated, not all inflammation is bad. And the experts say that individuals’ inflammatory response is very essential for the healing to effectively take place. It is this response that signals your body’s immune system to send some white blood cells and chemicals to help in fighting off the infection or repair any injury caused.

Finally it is worth noting that besides the known causes, there are other causes of chronic inflammation which may include obesity, smoking, stress, lack of exercise, exposure to secondhand smoke and diet choices. And to make the matter more worse, chronic inflammation in many cases does not show signs. Even though there will be no signs, there is the side which is good. And the good news is you can reduce chronic inflammation and lower your cancer risks. It starts with your diet. An anti-inflammatory diet also can help you avoid diabetes, heart disease and Alzheimer’s. Doctor Dalal Akoury is sharing with us some of the anti-inflammatory diet tips in our subsequent articles and so we want to encourage you to keep focused on the link for this great health piece of information. In the meantime you may want to consult with doctor Akoury for clarity in any concerns you may have over inflammation effects on cancer patients. You can call her on telephone number 843 213 1480 for further direction.

Inflammation effects on cancer patients: Benefits of good nutrition

http://www.awaremednetwork.com/

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Body inflammation causes obesity

Body inflammation causes obesity-How it happens

Inflammation

When inflammation becomes chronic, as is the case with obesity, chemical mediators, derived from different cellular activities, change in dynamics causing a progressive state of decline

A common theme that links many diseases and chronic illness is uncontrolled cellular inflammation. It is a factor in diseases including cardiovascular disease, diabetes, cancer, arthritis and many autoimmune-related conditions. Obesity has recently been added to this group of diseases as it is now known to present a low grade inflammatory response within many of the body’s tissues, which cause deleterious effects, often leading to the development of cardiovascular and metabolic disease. It is well known that being overweight is detrimental to one’s health, but until recently the known mechanisms were limited. Scientists over the last decade have started to unravel the mystery of why obesity leads to premature death. Although there is still much to learn, it is valuable to comprehend the known effects of chronic inflammation, as the prevalence of obesity continues to be a rising problem among the American population, particularly in children.

Inflammation and obesity-What is the meaning

Inflammation is, by design, a protective response leading to the repair of tissue. When inflammation becomes chronic, as is the case with obesity, chemical mediators, derived from different cellular activities, change in dynamics causing a progressive state of decline. Fat cells are now considered an immune organ that secretes numerous immune modulating chemicals. Visceral fat, in particular, is associated with the low grade inflammation that seems to be a contributing pathologic feature for metabolic disease through insulin resistance and the promotion of atherosclerotic build-up in circulatory vessels. When high levels of visceral fat are combined with physical inactivity, over nutrition, and advancement in age, the effect becomes more pronounced. Visceral fat is highly metabolic and contributes to cytokine hyperactivity. Adipokines secreted from fat tissue influence the metabolic process and contribute to proper function. The consequent low grade inflammation associated with obesity causes disturbance in the secretion and function of adipokines. Research has identified changes in adiponectin, leptin, and resistin that exhibit harmful effects upon the body in obese individuals. Adiponectin is an antiatherogenic agent, meaning it helps prevent the development of atheroschlerotic plaque in blood vessels and slows the progression of atherosclerosis in coronary vessels. It does this by acting directly upon the vessel wall, inhibiting adhesive molecules from contributing to plaque formation and acts as a blocking agent to the formation of foam cells. In the skeletal muscle and the liver, adiponectin serves to promote insulin sensitivity and a positive blood lipid profile. Visceral adiposity reduces adiponectin concentrations. Lowering the adiponectin concentrations lessens the cardio protective effect, leading to increased cardiovascular risk.   Leptin regulates energy metabolism and balance in conjunction with the brain’s hypothalamus. Leptin is currently being touted as having cardioprotective benfits among its others roles in metabolism Leptin concentrations adjust in response to obesity and contribute to insulin resistance. The changes in leptin concentration have also been recognized as a risk factor for coronary heart disease. Likewise increased resistin concentrations correlate with obesity related inflammation and may be associated with the initiation and progression of atherosclerotic lesions. Resistin also promotes insulin resistance, although the actual mechanism is not known. Insulin resistance due to adipokine dysfunction is further influenced by free fatty acids liberated directly into the liver from visceral fat tissue. Visceral fat releases chemicals and fatty acids into the portal system where they act on the connecting organs. The portal circulation system is a specialized network of blood vessels that connect the visceral organs to the liver.   The excess fat in portal circulation has detrimental effects on insulin action, which is worsened by sympathetic hyperactivity in response to obesity. Sympathetic hyperactivity causes heightened lypolytic action resulting in excess free fatty acids in the blood. These actions combined with beta cell hypersecretion and reduced insulin clearance resulting in hyperinsulemia, lead to early stage diabetes.

Inflammation-Interleukin-6

Interleukin-6 (IL-6) is possibly another factor associated with inflammatory detriment within the portal system. High levels of IL-6 are a marker for inflammation and vascular pathology. Obese subjects demonstrated a 50% greater portal vein IL-6 concentration, demonstrating, again, the profound effect visceral fat has on pathogenic indicators. Portal vein IL-6 correlates with systemic C-reactive protein concentrations. C-reactive protein is associated with cardio- and peripheral vascular disease. C-reactive protein and oxidative stress are now presumed to interact in the early inflammatory processes of atherosclerosis. This is significant for young obese individuals. Although more research is necessary for conclusive association, C-reactive protein may be a new risk factor for CAD in individuals under 25 years of age.

Inflammation-imbalances

inflammation

If your immune system and its ability to quell inflammation in your body are impaired, watch out. You are headed toward illness and premature …

The imbalance between increased inflammatory stimuli with a concurrent reduction in anti-inflammatory activity may be the foundation for the accelerated endothelial dysfunction and insulin resistance associated with obesity and the comorbid disorders of metabolic disease.   More research is needed to clearly delineate the particular relationships, but it seems evident that the low grade inflammation caused by obesity and visceral adiposity lead to the premature development of disease. This, more so than ever before, identifies the importance of weight management during the developmental years and ongoing efforts to control weight throughout one’s lifespan.   For individuals that are currently obese, there is still plenty of hope. Weight loss is related to reduction of oxidative stress and inflammation, and these beneficial effects likely translate into reduction of cardiovascular risk in obese individuals.   Likewise, exercise and dietary management, along with pharmacologic intervention can lead to atherosclerotic reversal in the earlier stages of CAD. Individuals with central adiposity, poor blood lipid profiles, hypertension, and/or insulin resistance should seek immediate professional assistance to prevent further health detriment.   The recent inclusion of obesity among chronic diseases makes Dr. Akoury of AWAREmed Health and Wellness Resource Center very resourceful for you. She will help you achieve optimal weight loss, the Dr. focus on Neuroendocrine Restoration (NER) to reinstate normality through realization of the oneness of Spirit, Mind, and Body, Unifying the threesome into ONE. What an opportunity, try and relieve yourself of this problem for good.

Body inflammation causes obesity-How it happens
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