How To Re-Energize The Mitochondria By Use Of Intravenous Therapy

September 2, 2014

How To Re-Energize The Mitochondria By Use Of Intravenous Therapy To Prepare For Easy Addiction Detoxification

Intravenous Therapy Damages and the dysfunctions in mitochondria is an important factor in a range of human diseases due to their influence in cell metabolism. Mitochondrial disorders often manifest in a number of ways such as neurological disorders, diabetes and endocrinopathy.

In other diseases, defects occur in nuclear genes leading to dysfunctions of the mitochondrial proteins. Diseases such as hereditary spastic paraplegia, Friedreich’ ataxia and Wilson’s disease also result because of this. These diseases are inherited as in the case to other genetic diseases.

Mitochondria are central to oxidative phosphorylation and plenty of metabolism in the body and are also involved in plenty of cell deaths. Mitochondrial dysfunction therefore contributes to a wide range of human illnesses. Many patients with disease of the mitochondria also have issues with their gastro-intestinal tract and this is treated by therapies through nutritional intake. Such like patients usually have weak immune systems and as such they are always in constant battle with numerous infections. History and research reveals that earlier intravenous nutrition consisted of protein and dextrose alone. Lipids were then later introduced and later removed due to complications, but this was later re-introduced again.

At the moment or rather current time, there are multiple options in intravenous therapy for nutrition. The pump options are such as Gemstar and even CADD. Obtaining nutrients orally or through the gastro-intestinal is usually the most effective, since this is always a more physiological process.

However, if this is not possible, the intravenous nutrition is done, in the form of intravenous hydration or through home parenteral nutrition. Parenteral nutrition involves providing the patient’s calories using an intravenous line that leads into the central circulatory system. Intravenous nutrition involves giving fluids and can be used as a medium of providing electrolytes and other additives.

Various factors are important in determining the kind of hydration that is ideal for a patient. The length of time the procedure takes as well as how and where the hydration happens are most often the main factors to consider.

Hydration is a major issue with people suffering from mitochondrial disease. For one to decide to start intravenous hydration he should keep in mind whether the treatment is long-term or for a short term. The kind of lines through which intravenous hydration is done are peripheral, PICC line, PortACath and tunnel catheters.

Peripheral is placing the intravenous lines in the hand or arm and for a short period of time, normally between three to five days. This method is not ideal for use at home because it requires a professional to do it and fewer nurses have such kind of professional training.

Mitochondria PICC is placed with the aid of an X-ray in the upper arm where the catheter goes directly into the central circulatory system. The site or area of insertion should be protected from infections by always keeping it dry.

PortACath is usually placed in the upper chest of the patient.in this type of line, the needle can be removed and the patient is allowed to swim or even shower.

Tunnel catheters are also usually in the upper chest and surgically removed .however, they can also be placed somewhere hidden. There is a risk of infection with this type of intravenous hydration.

Many patients are allergic to the substances involved and suffer from skin breakdown and are therefore more susceptible to infections. Also because of lower immunity, they are prone to infections. Furthermore, these patients also stand a higher risk of clotting. These treatment lines should be handled with care and fewer people. They should be thoroughly washed and items cleaned separately. The access points should be sanitized with alcohol prior to injection.

Some complications such as catheter occlusion caused by blood clotting occur, due to thrombotic clots. But there are medications that clear clots on the catheter.

Infections are normally common because of a component in blood that encourages growth of bacteria. This component is known as biofilm. The biggest threat to intravenous nutrition however is catheter related blood stream infections. This is treated by antibiotics, antibiotic locks and by changing of the central line.

Intravenous therapy is also a great way to get a high dose of vitamins, vital minerals and antioxidants into the circulatory system. Therefore, the nutrients are quickly introduced into the circulation and are delivered directly into the cellular space without having to go through the gastro-intestinal tract.

Many people notice improved immunity, higher energy levels and enhanced detoxification through therapies.

Intra venous nutrient therapy also provides higher doses as compared to oral therapy.

Scientists have been studying the effects of higher doses of Vitamin C for quite a long time.

Vitamin C is essential as a water-soluble vitamin that is commonly also known as ascorbic acid. The antioxidant effects of Vitamin C have been researched extensively to show positive and protective benefits against many ailments.

Myer’s Cocktail, AMP/NAD Energy Shots and MSM Infusions can also be used to boost the mitochondria

One may just feel a cold or a viral illness is coming on but once you have an intravenous cocktail that may well restore your health.

Whether you need help on balancing adrenals or you are an addict who is looking for a natural way of gradually recovering from an addiction, Awaremed Resource And Wellness Center is your perfect solution. You are free to call us, for more go to www.awaremednetwork.com

How To Re-Energize The Mitochondria By Use Of Intravenous Therapy To Prepare For Easy Addiction Detoxification

Mitochondrial Restoration and Cancer Treatment

July 5, 2014

How Mitochondrial Restoration is Essential for Cancer Treatment

Cancer diagnosis has gradually become a worrying threat to patients all over the globe with the number of those diagnosed with the killer disease increasing every decade. Statistics in countries such as the US, India, and Canada which have unsuccessfully devoted a lot of resources in researching for the ultimate cancer cure show that millions of people are diagnosed with cancer every year despite the massive technological advancements.

Cancer has by and by grown into a global epidemic of overwhelming proportions with the statistics in the early 1900s showing that one in 20 people developed cancer, ‘40s one in 16 people developed cancer, in the 1970s the number was one in 10 while today it is one in three!

The overall death rates are falling in spite of the alarming rates of cancer incidences as a result of the so called 40-year ‘war on cancer’. Awareness creation on cancer has been farce. A deeper understanding on the same has been achieved that comprehensively have reduced its mortality cases worldwide.

Increased medical research on cancer in the recent decades have revealed how mitochondria play a central role in the propagation of the disease given the necessary conditions. As such, mitochondria have become key organelles involved by researchers and therapists in chemotherapy-induced apoptosis.

Researchers have given priority to the relationship between mitochondria, Reactive Oxygen Species signaling and activation of survival pathways under hypoxic conditions as the core subject of increased study. There is speculation that insights into the mechanisms involved in ROS signaling may offer ways of facilitating discovery specific cancer therapies.

These studies have noted that chemicals, viruses and various radiations are responsible for inducing mutations in genes encoding cancer causing proteins which in turn give rise to the disease.

A number of interrelated processes are involved in the formation of cancer in this manner, in which mitochondria, the ‘cellular power plants’ involved in cellular differentiation, signaling, cell death as well as the control of cell growth and cycle; play a central role.

The condition where parts of the body are deprived of oxygen (hypoxia) as well as stressful conditions of nutrient deprivation especially lack of glucose have been identified to promote the process of malignant transformation when a low percentage of cells maneuver and overcome cellular senescence through certain mutations.

Studies have identified cellular stress in the form of hypoxic as well as glycemic conditions as two of the major triggering events for cancer. Oncoproteins, or proteins encoded by oncogenes which have the potential to cause the transformation of a cell into a tumor cell if introduced into it, usually reinforce the process of cancer formation by altering gene expression to bring about a common set of changes in mitochondrial function and activity in cancer cells.

The cancer cells follow a program that do not conform to the normal cell behavior. The cell proliferation that is not regulated leads to formation of cellular masses that extend beyond the resting vasculature and this results in oxygen and nutrient deprivation.

The resulting condition of hypoxia then triggers a number of critical adaptations that enable cancer cells to survive. In this condition the process of apoptosis is suppressed and the glucose metabolism is altered.

Hypoxia causes elevation in mitochondrial production of chemically reactive molecules containing oxygen known as reactive oxygen species or ROS.

Scientific investigations over the recent decades suggest that oxygen depletion thus stimulates mitochondria to compensate increased reactive oxygen species (ROS). This activates signaling pathways that promotes cancer cells survival and the growth of the tumor in a condition where the cells that escape senescent elimination emerge with oncogenic mutations and become immortalized tumors.

In this regard, studies have established that directing attention to the mitochondrial dysfunction responsible for the particular cancerous condition would be a lot effective in treating cancer. These mitochondrial disorders may be caused by mutations as already mentioned or acquired or inherited in the mitochondrial DNA as well as in nuclear genes that provide coding for mitochondrial components.

Mitochondrial Restoration

An understanding of the exact nature of the disorder in question is a key prerequisite to establishing an accurate therapeutic approach to the particular mitochondrial dysfunction and subsequent treatment to cancer resulting from the same.

Scientists suggest the ATP Profile as the recommended test in diagnosing a number of the mitochondrial disorders, though research into this test is still underway to establish it as fully workable. It involves blood assessment at several levels which include the adenosine triphosphate (ATP) – the primary form of energy in the body and generated by the mitochondria.

The intermediate form of energy in the body also found in the blood, Adenosine Diphosphate (ADP) is also included in the test. This is used by mitochondria to make ATP. Studies have shown that the more severe the dysfunction the more severe the symptoms. With the results from the diagnosis, a number of supplements can be used to remedy the disorder with specific recommendations from the doctor. A common example that various doctors recommend is riboflavin or vitamin B2 for correcting mitochondrial problems. This should be accompanied with physical therapy that help improve range of motion and dexterity.

How Mitochondrial Restoration is Essential for Cancer Treatment

Tag Archives: Mitochondrial DNA