Category Archives: Heavy Metal Toxicit

Hyperbaric Oxygen Therapy (Hbot) Helps In Addiction Recovery

July 25, 2015

How Hyperbaric Oxygen Therapy (Hbot) Helps In Addiction Recovery

Hyperbaric Oxygen Therapy (HBOT) was reportedly used to treat 340 patients with severe cases of alcoholism, drug addiction, and narcomania during the post-intoxication periods. Out of the 340, 223 were alcoholics, 68 were drug addicts, while 49 were hard narcotic users. This is according to various medical reports released a decade after the new millennium. These figures no doubt brought a new sense of hope to many drug users, especially those on various drug addiction recovery programs and their physicians alike about the so called HBOT. Any wild guesses would suggest naturally they did ask the question; so what about it? Well..

Hyperbaric oxygen therapy (HBOT) is a natural healing process of the body through 100% oxygen inhalation. HBOT involves taking in pure oxygen under increased and controlled atmospheric pressure. It is an established treatment for decompression sickness and hazardous effects of deep-sea diving among others. Most notable of the other medical conditions where HBOT may be recommended include serious infections, air bubbles in the blood vessels, stubborn wounds that come about as a result of diabetes or radiation related injuries as well as long term treatment option for drug addiction.

Brief History of Hyperbaric Oxygen Therapy (HBOT); And How it Works

History of hyperbaric oxygen therapy dates back to 350 years ago. The first (HBOT) chamber was erected in the year 1662. Use of HBOT however properly began mid in the 18th C. It was first tested by the military in the 1930s, precisely to find out its significance in deep-sea diving and aeronautics. Serious studies however began later in the 1960s against a number of medical conditions. These activities have continued to date but more important is the fact HBOT has been found to have long term positive effects on addiction recovery. Check out this: www.awaremednetwork.com for the timelines.

Hyperbaric Oxygen Therapy

Atmospheric pressure at sea level is 1 ATM. People take in approximately 21% oxygen at the sea level. In a hyperbaric chamber however they breathe in 100% oxygen, usually because the pressure is increased to about 3 times that at sea level. Blood vessels therefore carry more oxygen, providing about 15-25 times more oxygen to the rest of the body organs and tissues. Oxygen is said to have abilities to heal naturally. Increasing volume of oxygen supply therefore promotes faster healing of various medical conditions as earlier mentioned. This always comes with a number of health benefits.

Effects of HBOT on Patients’ Health

Hyperbaric oxygen therapy treatment has a positive effect on the patient’s physical and psychological status during treatment sessions and could as well persist for a given period of time after treatment depending on the duration of exposure. Those with co-current symptoms and other initial status often experience sedative and bio-energizing effects. Various psychopathological examinations indicated the patients experienced faster decline of mental disorders, which brought about a further reduction in the duration of time spent while getting relevant treatment. Also, the parameters of central hemodynamics stabilized and myocardial status recorded further improvement. Such favorable attribute of HBOT were largely thought to be as a result of the anti-hypoxic detoxifying and bio-energetic effects of HBOT Treatments.

Normally, oxygen is supplied via red blood cells. Under intense pressure however oxygen dissolves into all other body’s parts and fluids including; the plasma, lymph, and into the bone and transported to areas with low oxygen uptake, normally because of damaged body cells or the blood vessels. This extra oxygen reach to all the body parts, including the damaged parts therefore is essential in aiding enables the body parts to develop its own healing mechanisms. Increased amount of oxygen also greatly boosts the white blood cells to kill bacteria even faster, thereby reducing swellings and rapid formation of new blood vessels in the affected areas.

Hyperbaric Oxygen Therapy

Hyperbaric Oxygen Therapy on Addiction Recovery

Increased levels of oxygen supply to the brain can also improve the brain’s ability to function. This is particularly significant in addiction recovery because as indicated hyperbaric oxygen therapy helps in neuroplasticity. This is the brain’s ability to recover from damage. Addiction distorts normal neuron pathways, that is, drugs often lead to formation of new neuron pathways that identifies and recognizes the substance. Since the brain has a natural recovery mechanism, and that HBOT has the ability to increase that mechanism, it is therefore a great way of treating drug addiction.

Other than recovering from brain damage, many addicts on rehabilitation programs where this particular method of treatment is used confess they become more alert and that they are able to think much more clearly during or after treatment. Effect of the same on the patients’ physical strengths is substantial, and also they exhibit healthier skins. In addition, many of these patients claim they feel healthier at the end of their sessions than when they start.

In order to achieve better results, many physicians have also reported combining hyperbaric oxygen therapy with amino acid therapy. While further researching on the possibilities of using HBOT in addiction treatment Dr. Kenneth Blum discovered two amino acid formulations, which were; “Mental Clarity and KB 220Z”. Later he said what he and his colleagues found were that the Mental Clarity component improved rational functions while KB 220Z calmed strong cravings addicts have for drugs.

Finally as mentioned earlier in this article, HBOT has had a long history with addiction treatment. Most of that has been controversial. Please sign up for this year’s August integrative addiction conference to get up to date information about HBOT. Visit http://www.integrativeaddiction2015.com. The link also contains information about how you may sign and participate in the August Integrative Addiction Conference plus a full list of the speakers lined up for the event.

How Oxygen Therapy (Hbot) Helps In Addiction Recovery

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Managing Forms of Fear in Addiction Recovery

July 20, 2015

Forms Of Fear And Addiction And How To Manage

Ever found yourself at a scene of accident? Ever watched a horror movie? Well many of us have.

It is indeed dreadful. These two however brings out different kinds of fear in many of us. One is the kind you never want to experience ever. The other is the kind that despite the fact it holds our breaths and makes our hearts beat a little faster we still mole around and find some fun in them. Or who enjoys accident scenes; trust me not even the corps. How about anaconda, or the vampire diaries, aren’t they some of your favorites? The answer is obvious. Now back to our subject, fear and addiction.

One of the greatest concerns of an addict on rehab is fear. The following are some of the most common forms of fear among the addicts.

  1. Fear of recovery from addiction

As mentioned in many other reports, addiction is both rewarding and reinforcing. One major concern therefore is losing the very substances that gave them a sense of happiness and joy, losing their primary forms of pleasure and replacing the same with new ones that do not in essence give similar kind of pleasure quickly. Many addicts wonder whether really they are able to sustain recovery and their life after victory.

fear

  1. Fear of Failure to Recover

It has also been mentioned a number times recovery is a long journey. In the course of it, there are several pitfalls and failures. There are times of doubt; there are those times the addict thinks s/he set her/his goals so high. At which point they think they do not have what it takes. And they attempt to fall back due to fear and addiction.

  1. Fear of Victory over Addiction

Isn’t it ironical this comes after that of disappointment? Could be there are good reasons for this too. Naturally human beings are curious. They often wonder what it is on the other side. Usually they do not know. This develops into a form of fear similar to one of the most dreaded, fear of the unknown. Isn’t it true people fear walking in the dark? Similarly addicts may not know exactly what would happen once they recover. They may know the benefits yes, but they may not yet know a whole lot of other things including; how to deal with the past, and new identities.

  1. Fear of Denial by Family and Friends           

Many addicts are slow on coming to terms with reality. They fail to admit they have a problem. They go about recovery either discretely or normally and in total disregard of their situation. They find it hard in the face of mockery and or rejection by their old friends. It could be the ones who led them into the problem in the first place or the new recruits who are probably still blinded by the short-term benefits of addiction and are yet to face any real consequences. Many addicts fear being judged. However the worst form of fear under this category is the fear to seek support.

  1. Fear of Falling Back

There are never assurances to recovery. They are never clear cut treatment. These make this form of fear very real. Recovery demands a lot of commitment. Many addicts therefore fail to take any steps concerned the whole process might fail they shall be right back where they started, states of both fear and addiction.

The road leading to addiction is straight and smooth. Addiction is both rewarding and repetitive, literally making the ride fun. But there are consequences, most of the time dreadful. This reminds us the way back to recovery. Unlike the road down to addiction, the one back to sobriety is rough and bumpy. Sometimes it is so rocky we get stuck, other times it’s so muddy and slippery we get back down to addiction and wait for another season. Faced with both fear and addiction, it is important to be bold and take the right steps back to recovery. What then must we do to manage such fear and addiction?

  1. Based on Fear of Recovery

The first thing to do when faced with any form of fear is to recognize it. The other thing to do is to overcome. However the latter is never easy. It is advisable to take a step at a time. Also, it is advisable to share the same with a therapist who will be able to walk you through the steps. The other forum, rather one of the best is sign up in a support group where you will likely meet other addicts undergoing the process, probably harboring the same fears. Don’t they say always there is security in numbers?

Fear

  1. Based on Fear of Failure

It is simple, first recognize the fact there are failures along this path. Second everyone is unique; it might be a simple straight path as so and so. Once these facts have settled drop a perfectionist kind of attitude and go through the process. When faced with difficulties, share the same with the therapist and try another approach. Also, persistence is crucial in managing this form of fear and addiction.

  1. Based on Fear of Victory

How about try not resisting this kind of fear? How about identifying it and yet letting it be? Many however have found a secret in simply letting the future be and living in the present.

  1. Based on Fear of Denial

This form of fear demands great support by trusted family and friends. It is overcome by signing up into support groups.

  1. Based on Fear of Falling Back

This has been solved by the discovery of the brain’s ability to reorganize itself to a state of normal functioning, neuroplasticity. The secret here is in abstinence; this is so that the brain is able to remold back to a former state of sobriety. It also involves exposing yourself more to alternative forms of pleasure.

The above, very important, are a tip of the iceberg. There is a lot more to fear and addiction you may find very useful to you. Click on http://www.integrativeaddiction2015.com to find a whole piece of information on fear and addiction.

Forms Of Fear And Addiction And How To Manage

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Neuroplasticity In The Mesolimbic Dopamine System And Cocaine Addiction

July 16, 2015

Neuroplasticity In The Mesolimbic Dopamine System And Cocaine Addiction

Cocaine is the most addictive of all forms substance abuse. It is characterized by a high compulsion and relapse. Despite several years of clinical research, scientists are yet to find an effective medication. However some studies indicate the activity of neurons in the mesolimbic dopamine system, which comprises cells in the Ventral Tegmental Area (VTA) that develop into the medial and detour prefrontal cortex, amygdala, and accumbent, motivates cocaine reward thereby contributing to high compulsion.

Based on these research activities often called neuropharmacological studies, the addiction of cocaine is caused by neuroadaptations induced by the drug. This is so reportedly because of the learning, reward-related and memory processes of the mesolimbic dopamine systems’ circuitry where dopamine projections are developed.

Neuroadaptation Cause of Cocaine Compulsion

Neuroadaptations are understood to cause very high sensitivity to cocaine. They are also believed to cause hypersensitivity to cocaine-associated electrochemical signals such as irrational decision making and irregular cultured behaviors characterized by high insensitivity to dire consequences of addiction.           A major characteristics of cocaine addiction is its’s compulsive drug use despite adverse consequences and high rates of relapse during periods of abstinence. A current popular hypothesis is that compulsive cocaine use and cocaine relapse is due to drug-induced neuroadaptations in reward-related learning and memory processes, which cause hypersensitivity to cocaine-associated cues, impulsive decision making and abnormal habit-like learned behaviours that are insensitive to adverse consequences. Here, we review results from studies on the effect of cocaine exposure on selected signalling cascades, growth factors and physiological processes previously implicated in neuroplasticity underlying normal learning and memory. These include the extracellular signal-regulated kinase (ERK) signalling pathway, brain-derived neurotrophic factor (BDNF), glutamate transmission, and synaptic plasticity (primarily in the form of long-term potentiation and depression, LTP and LTD). We also discuss the degree to which these cocaine-induced neuroplasticity changes in the mesolimbic dopamine system mediate cocaine psychomotor sensitization and cocaine-seeking behaviours, as assessed in animal models of drug addiction. Finally, we speculate on how these factors may interact to initiate and sustain cocaine psychomotor sensitization and cocaine seeking.

mesolimbic dopamine system

The premise that cocaine has a neuroadaptation effect to the chemical composition of certain parts of the brain has motivated various studies on the part of cellular actions and signaling forces that altogether causes neuro-synaptic plasticity. Effects of long-term exposure to cocaine on signaling forces, growth elements, psychosocial and physiological processes of reward transmission initially linked to neuroplasticity as a cause of mental recovery are a substantial number. They include extracellular-controlled kinase, distortion of normal neuron pathways and other neurotrophic factors, neuro-synaptic plasticity, and glutamate factors.

Neuroplasticity in Mesolimbic Dopamine System  

Neuroplasticity is the brain’s ability to adjust to new environments or needs by developing new nerve cells throughout the body. It is the brain’s way of recovery. Neuroplasticity allows the cells to compensate for any injuries or diseases in the nerve system. It also allows the neurons reorganize themselves to perform new functions of the brain depending on changes in their working environment, also involves recovery from drug addiction such as that of cocaine.

Cocaine-induced neurochemical changes in glutamate transmissions and synaptic plasticity in the mesolimbic dopamine system facilitates cocaine psychomotor high sensitivity, compulsion, self-injection, and reinstatement, being interesting aspects of study in shedding light into cocaine addiction menace has been reviewed time and again.

Experimental Evidence of Neuroplasticity on Long-Term Exposure to Cocaine

A key consideration in the above reviews has been what experimental evidence are needed to derive a conclusion of the particular effects of long-term exposure to cocaine on neuroplasticity and how those effects facilitate the learned behavioral symptoms associated with that.

Given this objective, researchers made a strict condition that if so cocaine-induced neuroplasticity causes certain attributer learned behavior then a reversal of the physiological processes that led to that state should, therefore, guarantee a reduced exhibition of such behavior.

After further intense studies on the same, the condition is continuously being met. This has led to yet another attempt to evaluate the role of cocaine-induced neurochemical alterations in glutamate transmissions, synaptic plasticity in VTA, accumbens and amyglada in as earlier mentioned psychomotor hypersensitivity and compulsive behavioral characteristic of the drug.

Many of those studies found out repeated cocaine administration amplified the rate of activity of ERK in the development areas of the mesolimbic dopamine system, which includes the accumbens, amygdala and the prefrontal cortex of the brain.

ERK Phosphorylation in Mesolimbic Dopamine System

Triggers of increased ERK phosphorylation includes D1 dopamine receptors, (PKA) the dependent protein kinase and methyl-D-aspartic acid (NMDA). On the other hand it was observed triggers of reduced ERK phosphorylation include CREB the transcription factor, mitogen-and stress-activated protein kinase-1 (MSK-1), and immediate early genes Fos and Zif268.

Extracellular signal-controlled kinase activity and the subsequent ERK-mediated reduced gene transcription are crucial for increased cocaine-induced psych as a result of exposure to the drug. On the other hand increased cocaine-induced ERK activity in the mesolimbic dopamine system does not facilitate the development of psych after a considerable time of withdrawal. Injection of either SL327 or VTA therefore before cocaine administration lessens sensitized the drug-induced movement during experimental tests for expression of psychomotor sensitization if done some time after withdrawal.

mesolimbic dopamine system

Cocaine Psychomotor Sensitization

More recent reports indicated psychomotor cocaine sensitization after several weeks of withdrawal from the drug increased ERK2 activity. This was linked to increased acumen α-amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA), and the receptor’s (AMPAR’s) surface appearance. However, no increases in ERK2 activity nor AMPAR surface expressions were observed in the specimens that did not exhibit psychomotor cocaine sensitization even after repeated non-dependent cocaine exposure and after some time of withdrawal.

Acumen’s ERK rate of activity possibly serves two specific roles in facilitating rewarding effects of the psychostimulant in a CPP procedure. During CPP training, the accumben’s rate of activity mediates consolidation of the learned behavior between the drug’s unconditioned rewarding effects and the drug’s related context during the CPP testing, ERK movement mediates serious expression of cocaine’s other habituated responses.

Systemic SL327 inoculations before cocaine CPP training prevented cocaine-induced accumbens, ERK phosphorylation and the subsequent expression of cocaine CPP. PD98059 accumben injections are given either before or after CPP training sessions blocked subsequent amphetamine CPP expression.

The relevance of the above fascinating correlational findings of cocaine’s compulsive characteristic, its’ psychomotor sensitization, and the ERK phosphorylation in the mesolimbic dopamine system is, however, a subject for further scientific, clinical research. Please sign up for this year’s August Integrative Addiction Medicine Conference to learn more about the same. Click the following link to get your chance to participate in the event: http://www.integrativeaddiction2015.com.

Neuroplasticity In The Mesolimbic Dopamine System And Cocaine Addiction

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EPCs Role in Cardiovascular Diseases

June 4, 2015

Endothelial Progenitor Cells Role in Cardiovascular Diseases

EPCsCurrently, regenerative medicine is on focus with hopes that it can be used in treatment of cardiovascular diseases. The circulating endothelial progenitor cells have been shown to possess an ability to form mature endothelial cells that can be useful in the process of vascular repair as well as neoangiogenesis. In preclinical studies, it has been shown that the circulating endothelial progenitor cells (EPCs) have the potency for cardiovascular regeneration. With this said, it is god to admit that there is still a lot needed to be done in this area to show the effectiveness of the regenerative activities of these EPCs. Here we look at how the EPCs relate to cardiovascular diseases.

Endothelial Progenitor Cells are biomarkers of cardiovascular diseases

Just like other stem cells the circulating endothelial progenitor cells have the ability to proliferate, migrate, and differentiate into mature endothelial cells. First discovered from the human peripheral blood, the EPCs were shown to incorporate into sites of physiological or pathological neovascularization. Ever since the EPCs were discovered, a lot has improved in the understanding of the blood vessel formation. There is a growing body of evidence that EPCs helps in providing postnatal vasculogenesis mechanism for neovascularization and vascular remodeling.

These cells have also been shown to possess diverse physiological functions and helps in the recovery process of myocardial ischemia and infarction, limb ischemia, wound healing, atherosclerosis, endogenous endothelial repair, and tumor vascularization. Earlier clinical trials have proved that EPC therapy is safe and feasible for the treatment of cardiovascular diseases. Plus, the circulating EPCs are also considered as biomarkers for coronary and peripheral artery disease. In spite of the medicinal abilities of these EPCs, there is still a lot to be done in order for the mechanism by which these cells work can be fully understood and be applied.

Despite the haze that still clouds the mechanism with which the EPCs work, it has been shown that EPCs locating to damaged tissues and organs proceeding vascular regeneration do not only help in the formation of the neovasculature but also produce a variety of proangiogenic cytokines and growth factors thus promoting proliferation and migration of pre-existing Endothelial Cells and activating angiogenesis to contribute to vascular regeneration. it is suggested that the EPCs owes their ability to contribute to vascular regeneration to the presence of cytokines and other secreting pro-angiogenic factors in them such as VEGF, stroma derived factor (SDF)-1α, angiopoietin-1 (Ang-1), hepatic growth factor (HGF), insulin-like growth factor (IGF)-1, and eNOS/iNOS (inducible nitric oxide synthase). Thus, EPCs can mediate tissue-protective effects and contribute to neovascularization via direct vasculogenesis in ischemic tissues and indirect production of proangiogenic factors to pre-existing endothelial cells.

Research has shown that there are reduced numbers of EPCs in clinical conditions like diabetes mellitus, heart failure, chronic kidney disease and hypertension. In such conditions these EPCs also become impaired and malfunctioning. It has also been shown that the number of peripheral EPC is significantly reduced and their functions impaired in patients with established coronary artery disease and stroke. In contrast the number of EPCs has been found to be on the increase in patients with acute coronary syndrome such as acute myocardial infarction or unstable angina since they are mobilized from the bone marrow into the bloodstream.

The levels and migratory capacity of EPCs is also highly reduced in those suffering from hypertension with coronary artery disease. Besides, the concentration of circulating EPCs is considerably reduced in refractory hypertension as compared to healthy subjects. Imanishi T and his colleagues reported that EPC senescence is augmented in both experimental hypertensive rats and patients with essential hypertension, which may be related to telomerase inactivation. The team found that the hypertension-induced EPC senescence may affect the process of vascular remodeling.

Another researcher, Delva P in his research; Endothelial progenitor cells in patients with essential hypertension, reported that there was no alteration in the number or functional activity of EPCs in 36 patients with essential hypertension. Some research studies have also demonstrated that the numbers of EPCs are decreased in pulmonary hypertension while others that the number of EPCs are increased or remain normal in pulmonary hypertension. With these available literature that are somehow conflicting there isn’t a clear relationship between EPCs and hypertension

EPCs and Heart Failure

EPCsTo show the relationship between heart failure and the number of circulating EPCs, Valgimigli M and his counterparts tested the level of EPCs in patients suffering from heart failure and they discovered that EPC mobilization occurred in heart failure and showed a biphasic response, with elevation in early stages while depression in the advanced stages. The increased EPCs was shown as a replication of a functional bone marrow response to diffuse and severe endothelial damage during the early stages of heart failure but an additional and significant increase of tumor necrosis factor (TNF-α) counteracted and overwhelmed the elevation of EPC mobilization in advanced disease phases by exerting a possible suppressive effect on hemopoiesis.

conference

With the above findings, it is clear that there is a relationship between the EPCs and the cardiovascular diseases. For more information about bone marrow transplant and stem cell transplantation, visit www.awaremednetwork.com Dr. Dalal Akoury, a doctor with years of experience in integrative medicine will be able to help. Don’t hesitate to check on http://www.integrativeaddiction2015.com  and learn about the upcoming Integrative Addiction Conference 2015. The conference will dwell on unique approaches to telling symptoms of addiction and how to help addicts.

Endothelial Progenitor Cells Role in Cardiovascular Diseases

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Allogeneic Stem Cells For Neurodegenerative Diseases

June 4, 2015

Allogeneic Stem Cells Role in Neurodegenerative Diseases

stem cellThe stem cells are the precursors of all cells in the body. They are very essential for formation of new tissues and healing of damaged tissues as a result of diseases or even accidents. The stem cells are available in adequate quantities in the bone marrow and in the adipose tissues as well. They have the ability to turn into the red blood cells, platelets or even the white blood cells. They can also form the muscles cells, the bones and even cartilage and hence help in keeping your body healthy. naturally you should be having the healthy amounts of these stem cells, unfortunately due to diseases and accidents that may result in bone marrow destruction and damage you may have too little of these essential stem cells and so you will need a stem cell transplant. In most cases the stem cell transplant is done through an IV, supplementing the little stem cells you have left and hence enabling the body to work right.

When the stem cells are harvested from the same person, the transplant is termed as autologous stem cell transplant. the autologous stem cell transplant is the most popular as the cells are harvested from the patient’s own body and this rules out any chance of the transplant being rejected or causing any pathological reactions that may warranty serious medical attention. however there are cases when the stem cells can’t be sourced from the patient’s own body and this will dictate that the stem cells be sourced from a different person who is not genetically identical to the patient; this is termed as allogeneic stem cell transplant. For safety of any recipient in allogeneic stem cell transplant, it is important to match closely the immune system markers to reduce chances of any reaction that may inhibit the healing process.

Stem cells and neurodegenerative diseases

Neurodegenerative diseases are numerous and each has distinctive effects on the patient but all are caused as a result of neurodegeneration. Neurodegeneration refers to the damage of the nerves in that are essential for transmission of messages within the nervous system. When the nerve cells are destroyed and deformed, they lose their original structure they will begin to malfunction. In most cases this damage is done gradually but at the end of it all the patient will suffer from cognitive disabilities such as memory loss and poor decision making. There are numerous neurodegenerative diseases, however basing on their debilitating effects, just a few have been known and are being currently focused on. Some of the famous neurogenerative diseases includes; Huntington disease, Parkinson’s disease and Alzheimer’s disease. Apart from these diseases listed above there are others that have not been in the medical literature and are less publicized, this doesn’t mean that they are not worth the mention. They are.

The greatest danger of all neurodegenerative diseases is that they lead to progressive brain damage and neurodegeneration. The major three diseases may show different features and effects but they progress in similar manner at the cell level. take Parkinson’s disease for example, it affects the basal ganglia of the brain, depleting it of dopamine hence causing stiffness, rigidity and tremors in the major muscles of the body which are distinctive features of the disease.

In Alzheimer’s disease, it is characterized by deposits of minute protein plaques that damages different brain parts thereby leading to gradual memory loss while Huntington’s disease is known to corrupt the genetic make-up affecting muscles of the body thereby resulting in motor restriction and eventual death.

Allogeneic stem cells transplant

Allogeneic stem cellsThe cell is harvested from the bone marrow of a suitable donor by use of a needle. This maybe done repeatedly to draw the sufficient amounts needed for the transplant. After harvesting the cells from the marrow the blood is passed through a machine that separates the stem cells from the blood leaving the rest of the blood flowing back into the donor through the needle into the donors arm. The harvested stem cells can then be transplanted into the patient through a central venous catheter that is inserted into the patient’s chest. The stem cells flow through the catheter into the patient’s blood and into the bone marrow where they will give rise to other stem cells between one to three weeks.

After transplant the stem cells will begin to work in repairing and replacing the worn out tissues caused by various degenerative diseases. All this is made possible since they can form any cell in the body and help restore the normal body functions.

The neurodegenerative diseases have very debilitating effects; the inception of stem cell therapy seems to be a light at the end of the tunnel for those suffering from these diseases. Anyway you need to be updated on how well you can regain your health and vitality back even after suffering from these diseases.

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Get in contact with Dr. Dalal Akoury an experienced doctor in integrative medicine at www.awaremednetwork.com as well as http://www.integrativeaddiction2015.com and learn more about the upcoming Integrative Addiction Conference 2015. The conference will focus on the unique approaches to identifying symptoms of addiction and how to help addicts.

Allogeneic Stem Cells Role in Neurodegenerative Diseases

 

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