Weight loss and Toxic

March 14, 2014

Weight loss and Toxic-Need to detoxify

When making effort to lose weight, you may choose to look at your toxic level and try natural means

Many people are struggling with weight and obesity in the today’s society, in order for your body to lose weight, there must be adequate hormones insulin and leptin must be doing their work. Insulin is in place to control sugar and fat storage levels in the body while leptins job is the burning of fat. If these hormones and the mechanisms that are there to utilize them isn’t working, you will be unable to lose weight and extremely prone to packing it on. There are two reasons these hormones begin to fail in your body, the over-consumption of sugar and processed grains and toxicity.

Toxicity is perhaps the greatest problem in the fight to lose weight. When toxins enter your body they have an affinity for fat cells, due to the fact that they are fat soluble. When the toxins attach to the outer cell membrane which is made of a lipid bi-layer (two layers of fat), it causes the cells to continually release Leptin which is the hormone that tells the brain to burn fat for energy, therefore, weight gain that does not respond to exercise or diet is inevitably due to Leptin resistance.

The toxins also cause the release of chemicals called cytokines that damage Leptin receptors in the brain (hypothalamus). Once the receptors to Leptin have been damaged, Weight Loss becomes a problem your body then makes an extremely important hormone called MSH (Melanocyte Stimulating Hormone). MSH is produced in the hypothalamus by Leptin, and it controls nerve, hormone, cytokine functions, skin and mucus membrane defenses, as well as controls the production of endorphins and melatonin. If your brain cannot hear Leptin you will eventually become MSH deficient. The effect of this would be that every immune and hormone response in your body will be altered. This then leads to so many different diseases like depression, diabetes, cancer, heart disease, chronic fatigue, and fibro-myalgia, just to name a few.

In many ways, our polluted world is a true test of genetic survival of the fittest. The number of toxic chemicals now threatens the reproductive ability of the human race and is also a large part of the cancer issue. These chemicals contribute to weight gain in various ways, including disruption of the hormone signaling system that regulates your metabolism, damage to and accumulation in your white adipose tissue, and increased risk for poisoning during weight loss. It is absolutely vital that you understand this subject.

Weight loss and Toxic-Challenges

It is very clear that too toxins are released back into the circulation during weight loss. This is especially the case during significant weight loss. During a weight loss of 12 percent of body weight toxins in the blood increased 23 percent – 51 percent, with the heaviest individuals releasing the most toxins. Over a one year period of weight loss toxic exposure ranged up to a whopping 388 percent. Scientists have shown that such toxins can interfere with thyroid hormone function during weight loss. Human data shows that as the toxins go up in the blood during weight loss the levels of biologically active thyroid hormone (T3) go down. This data means that your plumbing and detoxification systems must be in good working condition for healthy weight loss or possibly even to engage weight loss.

Toxins make you feel irritable. Many people report feeling poisoned at a certain point in their weight loss process. Such people will always feel better when they eat a lot of food, as the toxins are pulled out of their blood and placed back in fat along with plenty of fat. With little effort, most people can readily lose weight they have most recently gained. After that, people reach what I will call the “toxic plateau.” This means that detoxification strategies may need to be adjusted if weight loss slows too much or stops.

Weight loss and Toxic-Toxic Environment

Once upon a time, the Environmental Protection Agency (EPA) conducted a program called the National Human Adipose Tissue Survey (NHATS). In 1982 and again in 1987 it analyzed human fat samples from cadavers obtained throughout the country, looking for the types of toxins that accumulate in human fat. Four industrial solvents and one dioxin were found in 100 percent of the fat samples. Nine more chemicals, including three more dioxins and one furan were found in more than 90 percent of the fat samples. In general, 83 percent of the fat samples contained PCBs. U.S. researchers have confirmed the presence of multiple toxins in human fat associated with obesity risk. The EPA has confirmed the presence of these chemicals as pollution in the farm soil across America this can only mean that this problem will be with us for some time to come.

The scientific theory of how these chemicals cause weight gain and difficulty losing weight has now been established. They bind to gene signaling within white adipose tissue and induce new fat cells to form while simultaneously increasing inflammation. Oftentimes, the newly formed fat cells are themselves damaged by the toxins so that they cannot metabolically perform, which includes an inability to make leptin normally. These damaged fat cells can fill up with excess fat and toxins, but are not able to efficiently carry out normal functions of fat cells, leading directly to increased risk for type 2 diabetes via the suppression of the important fat cell hormone known as adiponectin. Several human studies confirm that PCBs increase diabetes risk. These chemicals pose a serious problem to the thyroid gland and the efficient utilization of thyroid hormone throughout your body.

Weight loss and Toxic-Strategies detoxification during Weight Loss

The steps to take to boost antioxidant nutrients that are known to help detoxification processes while protecting the liver, brain, white adipose tissue, and/or circulation. Top choices are silymarin, R-alpha lipoic acid, chlorella, quercetin, grape seed extract and tocotrienols. It is now understood that “toxic” blood triggers the formation of new fat cells. This is because the endothelial cells of your circulatory system directly communicate to your baby fat cells and can tell them what to do. Keeping your blood as clean as possible and supporting your liver are vital steps to ensure that this process goes smoothly. While making all these efforts a decision to visit AWAREmed Health and Wellness Resource Center under Doctor Akoury’s who cares a lot will make great difference here we focus on Neuroendocrine Restoration (NER) to reinstate normality through realization of the oneness of Spirit, Mind, and Body, Unifying the threesome into ONE.

Weight loss and Toxic-Need to detoxify

Obesity and sugar addiction

March 13, 2014

Obesity and sugar addiction-Neuroendocrine imbalances

Obesity and sugar addiction-Sugar, sweetness or palatability

Sugar addiction is associated with obesity what you eat can have diverse effect on your life.

The concept of sugar addiction relies on rats given the choice between a palatable sucrose solution and a much less palatable chow. Naturally in such circumstances they consume sucrose. The question is whether it is sucrose, sweetness or palatability to which they are responding? It needs to be demonstrated that similar behavior could not be demonstrated with carbohydrate in general, artificial sweeteners or fat-rich palatable foods. Comparisons have been made between the reaction of rats to the provision of sucrose, a high-fat diet and a sweet-fat combination. The ability of the opioid antagonist naloxone to produce withdrawal symptoms was not observed with fat although it occurred when sucrose alone was provided, evidence that in this paradigm different types of palatable food produce different responses.

However, it appears that rats do not have a preference for sucrose consumption as there is a preference for sucrose in sham feeding studies, where after passing through the mouth it leaves the body, ensuring no post-ingestive effects occur. Dopamine is released from the nucleus accumbency with this procedure. The sweetness of fruit juices is rewarding as judged by “reward expectation-related neuronal activity” in the primate striatum, although it is produced by sugars other than sucrose.

There is a preference for artificial sweeteners that in turn have been shown to influence the activity of the nucleus accumbency. The intermittent access of rats to a saccharin solution rather than sucrose has also resulted in withdrawal symptoms when consumption stopped. It appears that in part at least there is a response not to sucrose but rather to a sweet taste.

More generally, is the response of a rat specifically to sweetness rather than palatability? Woolley questioned whether the opioid regulation of food consumption reflects the macronutrient content rather than flavor. They studied the consumption of two types of food pellets that differed in flavor although they were nutritionally identical. A μ-opioid receptor agonist injected into the nucleus accumbency increased the consumption of both pellets in a similar manner if they were tested when only one of the two foods was present. However, when both flavors of pellets were presented simultaneously, the agonist increased and the antagonist naltrexone selectively decreased the consumption of the preferred flavor. The authors suggested that based exclusively on flavor cues, opioid mechanisms in the nucleus accumbency increase the intake of palatable foods. Similarly the administration of naltrexone into the nucleus accumbency selectively decreased sucrose intake, although it had only a minimal influence on the consumption of less preferred chow. In addition a specific μ-agonist selectively increased the intake of sucrose, saccharin and a dilute saline solution. These findings demonstrated an important role for opioids in the nucleus accumbency in promoting the consumption of preferred palatable foods. When rats consumed a high-palatability sucrose solution the release of dopamine in the nucleus accumbency was dose dependent but palatable high-fat/sweet foods similarly induced dopamine release. The message is that it is palatability rather than sweetness or being sucrose that is critical in determining food preference.

This conclusion is supported by studying the impact of opioid drugs. As a generalization it has been known for many years that opioid agents enhance and opioid antagonists decrease feeding. In the rat the positive facial response to a sucrose solution was enhanced by morphine and decreased by opioid antagonists. The administration of morphine caused a short-term increase in food intake, and at least initially an increase in fat intake at the expense of carbohydrate.

The opioid antagonist, naloxone decreased fat rather than carbohydrate consumption in rats. As it is known that for many rats fat is more attractive than carbohydrates these finding are consistent with the view that opioid mechanisms influence the intake of palatable foods. Such a suggestion is supported by the study of initial food preferences. As there is variability amongst rats in their preferences for carbohydrate and fat Gosnell considered whether morphine was acting on food preferences. They distinguished fat-preferring from carbohydrate-preferring rats. Morphine increased carbohydrate intake in carbohydrate-preferring rats and increased fat intake in fat-preferring rats. Therefore morphine increased the intake of the preferred diet rather than a specific macronutrient. Similarly naloxone selectively decreased the intake of preferred foods and not sucrose content as would be predicted by the ‘sugar addiction’ hypothesis.

Obesity and sugar addiction-Sugar and reward mechanisms

Addictive drugs and palatable food both release dopamine from the nucleus accumbency.

The nucleus accumbency has different populations of neurons that are activated by natural and drug reinforcement. The release of dopamine by natural rewards, unlike drugs of abuse, undergoes rapid habituation.

Although the food-induced release of dopamine is markedly inhibited by pre-exposure to visual and olfactory stimuli that have been conditioned to food, similar visual and olfactory stimuli that had previously been conditioned to drugs of abuse strongly potentiate the dopaminergic reaction.

The suggestion, based on the animal evidence, is not that palatable foods are physically addictive but rather that a particular style of eating can produce a reaction to food that is similar to the response to drugs of abuse.

Obesity and sugar addiction-Comparisons of dopamine release induced by food and drugs of abuse

Although addictive drugs and palatable food both increase the release of dopamine from the nucleus accumbency it appears that they influence different populations of neurons. Such a conclusion is supported by studies in which either pharmacological manipulation or selective lesions reduce the self-administration of cocaine but do not influence the response to natural rewards. For example Caine and Koob used 6-hydroxydopamine to deplete the nucleus accumbency of dopamine and found a reduction in cocaine self-administration without altering the response to food.

Additional evidence arises from the study of the time scale of dopamine release. Dopaminergic functioning can be estimated using a range of methods. Recording the rate of firing of dopaminergic neurons allows the examination of functioning in a milli-second time frame. Similarly voltammetry measures dopamine release over sub-second periods. In contrast, microdialysis is used to estimate extra-cellular concentrations of dopamine over longer periods.

Finally research and studies will always be done on this topic obesity and addiction to find the scientific solution, nevertheless to achieve optimal weight loss in a record time efficiently and economically, it will be necessary to pay a visit to AWAREmed Health and Wellness Resource Center under Doctor Akoury’s where a lot of focus is put on Neuroendocrine Restoration (NER) to reinstate normality through realization of the oneness of Spirit, Mind, and Body, Unifying the threesome into ONE. This way you will get help fast while the endless researches and studies continue.

Obesity and sugar addiction-Neuroendocrine imbalances

Obesity and emerging neurobiology addiction

March 12, 2014

Obesity and emerging neurobiology addiction-Food addiction

Emerging neurobiology addiction offers healthy ground for obesity take a walk swim your weight away at the beaches.

It’s believed that the increased availability and consumption of highly palatable foods is the major factor behind the rise of obesity in developed countries. Many of these highly palatable foods are sweet tasting and appealing foods that contain high levels of the natural sugar sucrose. There are alternatives to sucrose the artificial sweetener sucralose is 600 times sweeter and does not contain calories but the obesity epidemic continues because the society prefers sucrose to sucralose.

Progress has been made in recent years in understanding the neurobiological underpinnings for this preference, sucrose activates dopamine neurons in a section of the brain called the striatum, and the resulting release of dopamine is associated with pleasure. Sucralose, on the other hand, does not have this effect. Moreover, the repeated consumption of high levels of sucrose can create a cycle of continued overconsumption even compulsive eating in order to recapture the initial feelings of pleasure. This is similar in many ways to drug abuse or addiction, and also involves some of the same signaling pathways within the body.

Obesity and emerging neurobiology addiction-Optogenic techniques

Researches used optogenetic techniques to show that activating dopamine neurons in the brain encouraged mice to eat in the absence of a sweet taste. The mice preferred water to sugar if their dopamine neurons were activated at the same time that they were offered the water.

A team of researchers reports that the release of dopamine is driven by melanin-concentrating hormone (MCH) neurons in the lateral hypothalamus of the brain. The experiments relied on transgenic mice in which the MCH neurons could be activated with a carefully directed light stimulus, and showed that mice preferred sucralose to sucrose when their MCH neurons had been activated. This finding was confirmed with mutant mice that lacked the cellular machinery required to recognize sweet tastes; these mice still preferred sucrose over sucralose because of its caloric content.

Researchers then engineered mice that did not have any MCH neurons at all. The fact that these mice preferred sucralose to sucrose supports a model in which MCH neurons appear to function as both a sucrose sensor and a nutrient sensor. Taken together, the results demonstrate that preference for sucrose is dependent on at least three factors: its ability to activate MCH neurons; its sweet taste as detected by the taste buds; and, as previously reported, the metabolic state of the organism. Consistent with these findings, they propose a model in which MCH neurons act as a key component of the neuronal networks that lead to the release of dopamine in response to the detection of sucrose.

Obesity and emerging neurobiology addiction-Different responses of sucrose and sucralose

A researcher showed that sucrose activates melanin-concentrating hormone (MCH) neurons in the lateral hypothalamus (LH), which leads to the release of dopamine (DA) in the striatum: pathway shown in red. In contrast, sucralose can only induce the release of dopamine if optogenetic techniques are used to activate the MCH neurons; pathway shown in green. However, it is almost certain that various other neuronal pathways (shown by black dashed lines) are involved; these might include orexin/hypocretin (OX) neurons in the lateral hypothalamus, or other neurons elsewhere in the brain.

While this latest work shows that sucrose preference is based on its nutritive value, the actual molecular mechanisms via which it affects MCH neurons remains unknown. According to one report, since sucrose contains glucose, it could modulate the excitability of MCH neurons via potassium channels that are gated by ATP.

Experts now suggest that the activation of the MCH neurons by sucrose might be mediated indirectly by afferent nerve fibers originating from taste sensors in the taste buds and/or the gastrointestinal tract. The receptors that respond to sweet tastes also respond to the nutritional content of the food and help determine which neuropeptides should be released by taste buds Moreover, sucrose, unlike sucralose, is known to act on taste receptors in the gastrointestinal tract): this modulates the release of hormones which serve to inform the brain about the availability of calories in the body)

Taken together, these data suggest that taste receptors in both the gustatory system (the part of the sensory system that responds to taste) and the gastrointestinal system may respond to natural sugars in one way and to artificial sugars in a different way.

An outstanding challenge is to find out how MCH neurons activate dopamine neurons in the striatum. One possibility is that the orexin/hypocretin neurons in the hypothalamus have a role. Since these neurons are involved in the regulation of the desire for sugar, and they are also known to interact with MCH neurons, they might work together with MCH neurons to trigger reward.

The work of researchers represents a significant advance in our understanding of the neural mechanisms underlying how mammals respond to some kinds of foods. We now understand better why we have an innate desire for sweet foods, which are highly caloric and might have been, in the past, advantageous from an evolutionary perspective. Yet in the modern world, where highly caloric food is readily available, how do we resist this drive so as to avoid the many problems that are associated with obesity?

Interestingly, it appears that the increased use of non-caloric sugar substitutes as a mechanism to prevent weight gain or enhance weight loss has come at a cost. Recent studies show that prolonged consumption of sucralose and other high-intensity sweeteners can have potentially harmful effects on energy metabolism. On the other hand, it has been reported that non-caloric sugar substitutes do little to reduce feelings of hunger.

Noting the struggle of addiction, we need to consider using new methods of treatment that will not be addictive in anyway. For the most effective Addiction Recovery is not healthy to keep using the chemical medicine, but instead to use a Natural addiction treatment to rid the victim from drug addiction. Experience a revolutionary nutritionally assisted detox program with Doctor Dalal Akoury at AWEREmed health and wellness resource centre. Here we focus on Neuroendocrine Restoration (NER) to reinstate normality through realization of the oneness of Spirit, Mind, and Body, Unifying the threesome into ONE.

Obesity and emerging neurobiology addiction-Food addiction

Excess estrogen and weight gain

March 6, 2014

Excess estrogen and weight gain-The role of Estrogen

excess estrogen can be a problem in your desire to lose weight

Progesterone and estrogen have opposing functions in your system. When it comes to weight gain estrogen causes your body to make and store fat while progesterone encourages your body to use fat for energy. While estrogen impairs blood sugar control and interferes with thyroid function, progesterone helps normalize blood sugar and facilitate thyroid activity. A nice balance between the two helps assure a healthy body and one of progesterone’s main functions is to balance estrogen levels.

A number of things help get you out of balance. First, not only does estrogen encourage fat production, but your fat cells manufacture estrogen. This means the more fat you have the more estrogen your body produces, and the more estrogen your body produces the more you will be prone to weight gain.

All of us are exposed to environmental toxins which imitates estrogen and fool our bodies into thinking we have even more of this hormone in our system than we do. Some sources of xenohormones include livestock fed estrogenic compounds to make them nice and fat before they end up at your grocery store. You may also get estrogenic drugs from your water supply due to all the women taking synthetic hormones in birth control pills and menopause drugs.

Lack of progesterone can also be hard on thyroid health and we know there is a strong connection between having a healthy thyroid and weight loss. According to experts women taking medication for low thyroid may find that they need less or no thyroid medications after using natural progesterone. If this is the case if you take thyroid medication, then you need to work closely with your doctor to adjust your medication as needed when you use natural progesterone.

Natural progesterone creams are available over the counter or by prescription from compounding pharmacies which can make up a cream designed just for you. You may need to find a physician familiar with natural remedies to assist you in this process. Conventional physicians may suggest a medication containing progestin or synthetic progesterone. However these are not the same as natural progesterone and have serious potential side effects.

Estrogen balance is essential for achieving and maintaining fat loss. In men and premenopausal women, too much estrogen a condition called estrogen dominance causes toxic fat gain, water retention, bloating, and a host of other health and wellness issues. In women, with stress and age there is a natural decline in testosterone and progesterone levels, leaving a relative excess of estrogen.

Excess estrogen and weight gain-What causes estrogen dominance?
There are only two ways to accumulate excess estrogen in the body: we either produce too much of it on our own, or acquire it from our environment or diet. Unfortunately, accumulating estrogen is not hard. We are constantly exposed to estrogen-like compounds in foods that contain toxic pesticides, herbicides, and growth hormones. Many of these toxins are known to cause weight gain, which serves to fuel the production of more estrogen from our own fat cells. More weight gain then leads to insulin resistance which increases the risk of estrogen dominance.

Pharmaceutical hormones such as those used in hormone-replacement therapy (HRT) or the birth control pill can also increase estrogen, whether we take them actively or absorb them when they make their way into our drinking water. We are living in a virtual sea of harmful estrogens, and researchers are only beginning to identify the extent of this exposure on health in humans and even other species.

Excess estrogen and weight gain -Signs and symptoms of estrogen dominance
A premenopausal woman with estrogen dominance is likely to have PMS, too much body fat around the hips and difficulty in losing weight. Perhaps you have a history of gallstones, varicose veins, uterine fibroids, cervical dysplasia, endometriosis, or ovarian cysts. In both sexes, estrogen dominance is thought to be responsible for many types of cancers. This particular hormone imbalance could be one of the leading causes of breast, uterine, and prostate cancer.

Excess estrogen and weight gain-How to correct estrogen dominance
we can be reduced the dominance of estrogen by among other means using these foods or specific habits:

Since the liver breaks down estrogen, alcohol consumption, drug use, a fatty liver, liver disease, and any other factor that impairs healthy liver function can spur an estrogen build-up. If you occasionally consume alcohol, always take a 1mg folic acid and a B-complex vitamin to reduce its negative effects.
Bacterial imbalance in the gut, and other problems that compromise digestion, interfere with the proper elimination of estrogen from the body via the digestive tract. Be sure to include a probiotic daily with 15 billion units. Store it in the fridge and take one or two capsules twice daily on an empty stomach.
Insoluble fiber also binds to excess estrogen in the digestive tract, which is then excreted by the body. A fiber supplement can also affect the composition of intestinal bacteria and reduce the build-up and re-absorption of free-floating estrogen. Good sources include wheat bran, corn bran, rice bran, the skins of fruits and vegetables (apples, pears, berries, tomatoes, eggplant, zucchini and carrots), nuts (especially almonds), seeds (particularly sunflower seeds), soybeans, dried beans, and whole-grain foods.
Choose organic dairy and meat products to reduce your exposure to hormone additives.
Consume weak phytoestrogenic foods such as pomegranate, flaxseeds, pears, apples, berries, organic non-GMO fermented soy, wheat germ, oats, and barley.
The body requires sufficient intake of zinc, magnesium, vitamin B6 and other essential nutrients, not only to support the breakdown and elimination of estrogen, but also to aid the function of enzymes responsible for the conversion of testosterone to estrogen. Be sure to add a high quality multivitamin supplement daily.
Avoid exposure to xenoestrogens from plastics, cosmetics, and the birth control pill.
Avoid unfermented soy products like tofu and soy milk.
As the body responds to high levels of stress, it “steals” progesterone to manufacture the stress hormone cortisol, often leaving a relative excess of estrogen. Stress management is essential.
Infrared sauna treatments are an excellent way to rid your body of estrogen.
Maintaining poor sleep habits cause a reduction in the hormone melatonin, which helps protect against estrogen dominance. Aim for seven to eight hours of sleep per night in a cool, dark room.

In all these efforts of trying to lose, for real optimal weight loss, a visit at AWAREmed Health and Wellness Resource Center under Doctor Akoury’s will be the best choice you can make in your desire to lose weight. Doctor Akoury cares and she focuses on Neuroendocrine Restoration (NER) to reinstate normality through realization of the oneness of Spirit, Mind, and Body, Unifying the threesome into ONE. What else can be better?

Excess estrogen and weight gain-The role of Estrogen

Yo-Yo Dieting

February 27, 2014

Yo-Yo Dieting

Yo-Yo Dieting-What is Weight Cycling?

Yo yo dieting occurs when one loses a significant amount of weight but eventually gains it back

Is losing weight one of your concerns? Then chances are that you have heard of yo yo dieting. Yo yo dieting occurs when one loses a significant amount of weight but eventually gains it back. It is also called weight cycling because of its nature of occurrence that is its cycle or repetitive pattern of weight loss and weight gain.

Causes behind Yo Yo Dieting

It will be important to be acquainted with the common causes of yo yo dieting to avoid the possible problem it may bring in your life. The yo yo effect is usually caused by weight loss plans that involve missing meals, fasting and crash diets that drastically lower your daily caloric intake. When the body is deprived of the calories it needs for energy, it will adapts to the change by slowing down your metabolism.

Metabolism is the process through which your body burns the food you eats in order to produce energy. Your metabolism may not get back to normal even when you resume your normal lifestyle. A slower metabolism means more weight gain. In some cases, not only will you gain back the weight you lose during the diet, but you may even gain more than before.

Yo Yo Dieting and Muscle Loss

Muscle loss is another effect that is associated with yo yo dieting. When your daily caloric intake becomes extremely low, your body tends to digest your muscle cells. Less muscle mass also contributes to slower metabolism. Instead of the slim and toned figure that you’re aiming for, you end up with a thinner but flabbier body.

The Effects of Yo Yo Dieting

If you’ve encountered yo yo dieting before then you’re probably familiar with its effects. It’s true that most crash diets and other rapid weight loss solutions can succeed in helping you shed pounds in a short time. However, it’s important to know that this weight loss is generally due to the loss of water weight and muscle mass. Aside from the fact that the weight loss is merely temporary, yo yo dieting has other negative effects. For instance, your failure to lose weight permanently may eventually impact your emotional health. This results in depression, stress and eating disorders.

Fluctuating weight can also result in a higher risk of developing cancer and heart disease. You might also suffer from losing hair and developing osteoporosis, among other health conditions. This is due to the fact that most crash diets are lacking in nutrients that are essential for healthy hair, bones and good health in general. Because of the low caloric intake, fatigue and difficulty in concentrating might also result from yo yo dieting.

Besides the above some studies suggest that weight cycling may increase the risk for certain health problems. These include high blood pressure, high cholesterol, and gallbladder disease. For adults who are not obese and do not have weight-related health problems, experts recommend maintaining a stable weight to avoid any potential health risks associated with weight cycling. Obese adults, however, should continue to try to achieve modest weight loss to improve overall health and reduce the risk of developing obesity-related diseases.

Losing and regaining weight may have a negative psychological effect if you let yourself become discouraged or depressed. Weight cycling should not be a reason to “feel like a failure.” Instead it is a reason to refocus on making long-term changes in your diet and level of physical activity to help you keep off the pounds you lose.

Weight cycling has not been proven to increase the amount of fat tissue in people who lose and regain weight. Researchers have found that after a weight cycle, those who return to their original weights have the same amount of fat and lean tissue (muscle) as they did prior to weight cycling. People who exercise during a weight cycle may actually gain muscle.

Some people are concerned that weight cycling can put more fat around their abdominal (stomach) area. People who tend to carry excess fat in the stomach area (apple-shaped), instead of in the hips, thighs, and buttocks (pear-shaped), are more likely to develop type two diabetes heart diseases, and high blood pressure. Studies have not found, however, that after a weight cycle, people have more fat around their stomachs than they did before weight cycling.

If I regain lost weight, won’t losing it again be even harder?

A person who repeatedly loses and gains weight should not have more trouble trying to reach and maintain a healthy weight than a person attempting to lose weight for the first time. Most studies show that weight cycling does not affect one’s metabolic rate. Based on these findings, weight cycling should not affect the success of future weight-loss efforts. Metabolism does, however, slow down as a person ages.

In addition, older people are often less physically active than when they were younger. Regardless of your age, making regular physical activity as well as healthy eating habits a part of your life will aid weight loss and improve health overall.

Preventing Yo Yo Dieting

If you don’t want to be among the many victims of yo yo dieting, then you need to boost your metabolism instead of slowing it down. These can be achieved by increasing your amount of physical activities. You should also look for ways to lose weight gradually and safely, because this produces a more effective and longer-lasting weight loss.

Finally getting information from different sources is very good because it makes you be enlighten and do the right thing. To help you get further helpful information on optimal weight loss, a visit to AWAREmed Health and Wellness Resource Center under Doctor Akoury’s will offer much needed solution. Doctor Dalal Akoury cares; she focuses on Neuroendocrine Restoration (NER) to reinstate normality through realization of the oneness of Spirit, Mind, and Body, Unifying the threesome into ONE

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